Home > Journals > Minerva Cardioangiologica > Past Issues > Minerva Cardioangiologica 2018 August;66(4) > Minerva Cardioangiologica 2018 August;66(4):442-51

CURRENT ISSUE
 

JOURNAL TOOLS

eTOC
To subscribe
Submit an article
Recommend to your librarian
 

ARTICLE TOOLS

Publication history
Reprints
Permissions
Cite this article as

 

REVIEW  STEMI: CURRENT KNOWLEDGE AND FUTURE PERSPECTIVES 

Minerva Cardioangiologica 2018 August;66(4):442-51

DOI: 10.23736/S0026-4725.18.04614-5

Copyright © 2018 EDIZIONI MINERVA MEDICA

language: English

How the functional assessment of culprit and non-culprit lesions may improve stratification and treatment of STEMI patients

Gianluca CAMPO 1, 2, 3 , Francesco GALLO 1, Giulia BUGANI 1, Rita PAVASINI 1, Massimo FINESCHI 4

1 Cardiovascular Institute, Ferrara University Hospital, Cona, Ferrara, Italy; 2 Maria Cecilia Hospital, GVM Care and Research, Ettore Sansavini Health Science Foundation, Cotignola, Ravenna, Italy; 3 Department of Cardiovascular Diseases, Santa Maria alle Scotte, University Hospital, Siena, Italy


PDF


Reperfusion therapy of the infarct-related artery (IRA) with primary percutaneous coronary intervention is the cornerstone for the treatment of patients with ST-elevation myocardial infarction (STEMI). However, up to 30% of STEMI patients present a multi-vessel coronary artery disease. Several methods are now available for the assessment of functional severity of a coronary stenosis both for IRA and non-culprit coronary lesions. The functional assessment of the IRA has mainly a prognostic implication in terms of major adverse cardiovascular events, recovery of left ventricular function and evaluation myocardial viability. Conversely, the functional assessment of the non-culprit coronary lesions has a fundamental role to guide staged revascularization. The aim of this review is to revise the most validated methods to perform the functional assessment of both culprit and non-culprit lesion in ST-elevation myocardial infarction.


KEY WORDS: Myocardial fractional flow reserve - Hyperemia - ST elevation myocardial infarction

top of page