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Medicina dello Sport 2018 June;71(2):314-22

DOI: 10.23736/S0025-7826.18.03222-2

Copyright © 2018 EDIZIONI MINERVA MEDICA

language: English, Italian

Postexercise hypotension and pressure reactivity after a swing session with kettlebell: a pilot study

Alexandre R. PIRES FERREIRA 1, 2, 3, Felipe J. MARTINS 1, 2 , Dihogo G. DE MATOS 2, Bruno F. CASTRO 4, Luan M. AZEVÊDO 3, 5, 6, Marzo E. DA SILVA-GRIGOLLETO 1, 5, Emerson PARDONO 1, 3

1 Department of Physical Education, Federal University of Sergipe - UFS, São Cristovão, Sergipe, Brazil; 2 Group of Studies and Research of Performance, Sport, Health and Paralympic Sports - GEPEPS, the Federal University of Sergipe - UFS, São Cristovão, Sergipe, Brazil; 3 Group of Study and Research in Physical Exercise - GEPEF, Federal University of Sergipe - UFS, São Cristovão, Sergipe, Brazil; 4 University Center Jorge Amado, UNIJORGE, Salvador, Bahia, Brazil; 5 Functional Training Group - FTG, Federal University of Sergipe - UFS, São Cristovão, Sergipe, Brazil; 6 Mauricio de Nassau College, Aracaju, Sergipe, Brazil


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BACKGROUND: To analyze the acute effect of a kettlebell exercise session on blood pressure reactivity (BPR) and postexercise hypotension (PEH) in young and normotensive individuals.
METHODS: Eight volunteers practicing kettlebell for at least 6 months were recruited. They performed two experimental sessions: 1) a kettlebell session (KS) that consisted of a standardized pre warm-up followed by 20 minutes of kettlebell swinging; and 2) a control session (CS) that had the same duration as the session with the kettlebell, but without physical exercise. Blood pressure (BP) assessment occurred before and after each of the experimental sessions and BPR was evaluated with a Cold Pressor Test. Statistical analysis consisted of analysis of variance for repeated measurements (2×8), with multiple comparisons between Bonferroni pairs. The statistical significance was set at P≤0.05.
RESULTS: The KS caused a significant increase in systolic BP immediately after the end of the intervention (P≤0.05), but did not provoke PEH when compared to the CS. However, KS promoted attenuation of BPR for systolic BP after the first minute of the Cold Pressor Test in comparison with the CS (113.9±13.2 mmHg vs. 118.4±13.8 mmHg, respectively; P≤0.05). The diastolic PA showed no difference between the sessions; however, qualitatively the KS resulted in lower systolic and diastolic BP values than CS.
CONCLUSIONS: We conclude that a KS session is insufficient to cause PEH; however, it is a feasible strategy to attenuate BPR for systolic BP in normotensive young people.


KEY WORDS: Atheletes - Exercise - Postexercise hypotension

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