Advanced Search

Home > Journals > Medicina dello Sport > Past Issues > Medicina dello Sport 2001 June;54(2) > Medicina dello Sport 2001 June;54(2):105-13

ISSUES AND ARTICLES   MOST READ   eTOC

CURRENT ISSUEMEDICINA DELLO SPORT

A Journal on Sports Medicine

Official Journal of the Italian Sports Medicine Federation
Indexed/Abstracted in: BIOSIS Previews, EMBASE, Science Citation Index Expanded (SciSearch), Scopus
Impact Factor 0,163

Frequency: Quarterly

ISSN 0025-7826

Online ISSN 1827-1863

 

Medicina dello Sport 2001 June;54(2):105-13

    FUNCTIONAL EVALUATION SECTION

Effect of training, detraining and deconditioning on VO2 slow component

Maione D. 1, Senaldi R. 2, Maietta P. 1, Maione A. 1, Drago E. 2, Tentoni C. 1

1 ISEF - Facoltà di Scienze motorie, Università degli Studi - Bologna;
2 Centro Interuniversitario di Studi e Ricerche in Medicina dello Sport, Istituto di Medicina dello Sport CONI-FMSI - Bologna

This study was designed to evaluate the effects on .VO2 slow component (.VO2sc) of a 7-week aerobic training and of a detraining of the same duration in 4 subjects (I group); and of 4 weeks deconditioning in 3 subjects (II group). The 7 subjects (22.1±0.9 years), physically active, have performed a ramping-test for detecting the 1st ventilatory threshold (Sv1) and maximal aerobic capacity, on which the constant load exercises wattage had selected, always supra-Sv1. These latter have been used for the .VO2 kinetics study, employing mathematical models with 2 exponential terms fitted on experimental data through an original software. The .VO2sc entity in O2 ml/kg has been obtained by the integral of the area among the 2 exponential of the mathematical model. As to deconditioning, the group II subjects have stopped each attivity in gym. In group I the .VO2sc in average, reduced with the training of ~ 42%, and increased after detraining of ~ 68%; between pretraining and detraining it decreased of 10%. In group II after deconditioning .VO2sc increased in average of ~ 22%- The .VO2sc reduction with training ad its increase with detraining confirm the results of previous studies. While the deconditioning has increased the .VO2sc, this is decreased, even a little, between pretraining and detraining. It’s likely that the detraining effects influence the .VO2sc responsible mechanisms in more extended times respect to those from us employed, subsequently to the loss of training-induced advantages.

language: Italian


FULL TEXT  REPRINTS

top of page